2025
GWAS meta-analysis identifies five susceptibility loci for endometrial cancer
Ramachandran D, Wang X, Laisk T, Zheng Y, Ingold N, Canson D, Kho P, Naumann B, Chapman C, Bousset K, Krause A, Schürmann P, Wieland B, Hanel P, Hülse F, Häfner N, Runnebaum I, Dubrowinskaja N, Turmanov N, Yugay T, Yessimsiitova Z, Amant F, Annibali D, Beckmann M, Bodelon C, Buchanan D, Chen C, Clarke M, Cook L, De Vivo I, De Wispelaere W, Du M, Easton D, Emons J, Fasching P, Friedenreich C, Gallagher G, Giles G, Goode E, Harris H, Hunter D, Kolin D, Kraft P, Lacey J, Lambrechts D, Lu L, Mutter G, Naduparambil J, O’Connell K, Patel A, Pharoah P, Rebbeck T, Ricceri F, Risch H, Ruebner M, Sacerdote C, Scott R, Setiawan V, Shu X, Southey M, Tham E, Tomlinson I, Turman C, Wentzensen N, Xu W, Yu H, Zheng W, Spurdle A, Yarden Y, Team E, Mägi R, Hillemanns P, Glubb D, Dörk T, O’Mara T. GWAS meta-analysis identifies five susceptibility loci for endometrial cancer. EBioMedicine 2025, 118: 105830. PMID: 40633141, PMCID: PMC12275056, DOI: 10.1016/j.ebiom.2025.105830.Peer-Reviewed Original ResearchConceptsGenome-wide association studiesGenome-wide association study meta-analysisRisk lociGenome-wide association study analysisGenome-wide significant risk lociTumor suppressorNeuron navigator 3Significant risk lociGene-based analysisEndometrial Cancer Association ConsortiumSignificant lociValidation genotypingAssociation studiesSusceptibility lociMeta-analysisEndometrial cancer casesCell divisionHigh-income countriesLociEnvironmental risk factorsCell deathCell survivalEndometrial cancerNAV3Case-control series
2018
A G3BP1-interacting lncRNA promotes ferroptosis and apoptosis in cancer via nuclear sequestration of p53
Mao C, Wang X, Liu Y, Wang M, Yan B, Jiang Y, Shi Y, Shen Y, Liu X, Lai W, Yang R, Xiao D, Cheng Y, Liu S, Zhou H, Cao Y, Yu W, Muegge K, Yu H, Tao Y. A G3BP1-interacting lncRNA promotes ferroptosis and apoptosis in cancer via nuclear sequestration of p53. Cancer Research 2018, 78: canres.3454.2017. PMID: 29588351, PMCID: PMC8073197, DOI: 10.1158/0008-5472.can-17-3454.Peer-Reviewed Original ResearchMeSH KeywordsAnimalsApoptosisBreast NeoplasmsCell Cycle CheckpointsCell NucleusCytoplasmDatasets as TopicDisease ProgressionDNA HelicasesDown-RegulationFemaleGene Expression Regulation, NeoplasticGenes, Tumor SuppressorHumansKaplan-Meier EstimateLung NeoplasmsMaleMiceMice, NudeMice, SCIDPoly-ADP-Ribose Binding ProteinsProtein BindingRNA HelicasesRNA Recognition MotifRNA Recognition Motif ProteinsRNA, Long NoncodingRNA, Small InterferingSignal TransductionTumor Suppressor Protein p53Xenograft Model Antitumor AssaysConceptsTumor suppressorCancer progressionProtein-binding protein 1Long noncoding RNACell cycle arrestLncRNA functionsRas GTPaseWild-type p53Interaction domainMetabolic genesNoncoding RNAsFunctional domainsP53 modulatorsNucleotides 1P53 pathwayProtein 1Precise roleTypes of cancerLncRNAsSuppressorFerroptosisApoptosisP53ExpressionCfp1
2016
Complement component 7 (C7), a potential tumor suppressor, is correlated with tumor progression and prognosis
Ying L, Zhang F, Pan X, Chen K, Zhang N, Jin J, Wu J, Feng J, Yu H, Jin H, Su D. Complement component 7 (C7), a potential tumor suppressor, is correlated with tumor progression and prognosis. Oncotarget 2016, 5: 86536-86546. PMID: 27852032, PMCID: PMC5349933, DOI: 10.18632/oncotarget.13294.Peer-Reviewed Original ResearchConceptsNon-small cell lung cancerComplement component 7NSCLC patientsPotential tumor suppressorOvarian cancerOvarian tissueMultivariate Cox regression analysisLow expressionZhejiang Cancer HospitalIndependent prognostic predictorCox regression analysisPrognosis of patientsAdvanced clinical stageCell lung cancerNormal ovarian tissuesExpression of C7Malignant ovarian tissuesTumor suppressorQuantitative polymerase chain reactionCancer HospitalClinical stagePrognostic predictorLung cancerPolymerase chain reactionPoor differentiation
This site is protected by hCaptcha and its Privacy Policy and Terms of Service apply